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For the cardiotoxic effects of sorafenib (SOR), the upregulation of ATF4 expression attenuated cardiomyocyte ferroptosis ( Increased ATF6 expression induced by androgen deprivation in prostate cancer renders prostate cancer (Pca) cells resistant to ferroptosis

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It requires selenium as a cofactor and contains a selenocysteine amino acid residue in the active site of each monomer that participates in the actual mechanism of the enzyme